| Nombre | MARIA SANDRA CABRERA BENITEZ |
|---|---|
| Área | BIOLOGÍA Y QUÍMICA |
| Campo | BIOLOGÍA Y QUÍMICA |
| Disciplina | BIOLOGÍA MOLECULAR |
| Especialidad | OTRAS |
| CVU | 40952 |
| Institución | |
|---|---|
| Dependencia | SECRETARIA GENERAL |
| Entidad | CIUDAD DE MEXICO |
| Nivel | 1 |
| Vigencia | Inicio: 01/01/2025 |
| Fin: 31/12/2029 |
Publicaciones ORCID
- 2026
- How experimental models have unlocked hidden mechanisms of hypersensitivity pneumonitis European Respiratory Review
- Autophagy impairment by ATG4B deficiency reduces experimental hypersensitivity pneumonitis severity
- 2024
- Increased ER Stress and Unfolded Protein Response Activation in Epithelial and Inflammatory Cells in Hypersensitivity Pneumonitis Journal of Histochemistry & Cytochemistry
- 2023
- Nucleophagy contributes to genome stability through degradation of type II topoisomerases A and B and nucleolar components Journal of Cell Science
- 2021
- Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1 Autophagy
- 2020
- Identification of Autophagy-related Proteins in Lungs From Hypersensitivity Pneumonitis Patients Journal of Histochemistry & Cytochemistry
- 2019
- Delayed resolution of bleomycin-induced pulmonary fibrosis in absence of MMP13 (collagenase 3). American journal of physiology. Lung cellular and molecular physiology
- 2018
- Impaired autophagic activity and ATG4B deficiency are associated with increased endoplasmic reticulum stress-induced lung injury. Aging
- Upregulation and Nuclear Location of MMP28 in Alveolar Epithelium of Idiopathic Pulmonary Fibrosis. American journal of respiratory cell and molecular biology
- 2016
- Role of matrix metalloproteinases in the pathogenesis of idiopathic pulmonary fibrosis. Respiratory research