Información*
| Nombre | DAVID SANTIAGO GERMAN |
|---|---|
| Área | MEDICINA Y CIENCIAS DE LA SALUD |
| Campo | ESPECIALIDAD MÉDICA |
| Disciplina | URGENCIAS MEDICAS |
| Especialidad | EMERGENCIAS |
| CVU | 277572 |
| Institución | |
|---|---|
| Dependencia | OOAD D.F. NORTE |
| Entidad | CIUDAD DE MEXICO |
| Nivel | 1 |
| Vigencia | Inicio: 01/01/2022 |
| Fin: 31/12/2027 |
* Información del primer trimestre de 2026.
Fuente: SECIHTI.
Publicaciones ORCID
- 2023
- Genetic Variants Associated with High Susceptibility of Premature Ischemic Stroke Journal of the Renin-Angiotensin-Aldosterone System
- Polymorphisms in the Renin-Angiotensin System and eNOS Glu298Asp Genes Are Associated with Increased Risk for Essential Hypertension in a Mexican Population Journal of the Renin-Angiotensin-Aldosterone System
- 2021
- Identification of genetic risk factors associated with ischaemic stroke in young Mexican patients,Identificación de factores de riesgo genéticos asociados a la enfermedad vascular cerebral de tipo isquémico en jóvenes mexicanos Neurologia
- 2020
- D-dimer as A Biomarker of Severity and Adverse Outcomes in Patients with Community Acquired Pneumonia Archives of Medical Research
- 2019
- Gene polymorphisms of angiotensin-converting enzyme and angiotensinogen and risk of idiopathic ischemic stroke Gene
- Genetic Polymorphisms Associated with Thrombotic Disease Comparison of Two Territories: Myocardial Infarction and Ischemic Stroke Disease Markers
- 2018
- Association of renin-angiotensin system genes polymorphisms and risk of premature ST elevation myocardial infarction in young Mexican population Blood Coagulation and Fibrinolysis
- Polimorfismos asociados a disfunción endotelial y a un estado protrombótico en jóvenes mexicanos con infarto cerebral Gaceta medica de Mexico
- 2017
- Hypofibrinolytic State in Subjects with Type 2 Diabetes Mellitus Aggravated by the Metabolic Syndrome before Clinical Manifestations of Atherothrombotic Disease BioMed Research International
- Reduced proliferation of endothelial colony-forming cells in unprovoked venous thromboembolic disease as a consequence of endothelial dysfunction PLoS ONE